By Elmer H. Marth, James Steele

ISBN-10: 082470536X

ISBN-13: 9780824705367

This completely revised and up to date reference/text presents accomplished insurance of the newest advancements and clinical advances in dairy microbiology. It emphasizes probiotics, fermented dairy items, affliction prevention, and public future health and regulatory keep an eye on criteria for dairy foods.Additionally, utilized Dairy Microbiology, moment version provides new chapters at the use, metabolism, and genetics of starter cultures, together with fresh information on isolation and enumeration of lactic acid micro organism.

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Feeding of starches that induce lactic acidosis). Numerous proposals have been put forward to ‘‘improve’’ the ruminal fermentation. These proposals have aimed at one or more objectives: (a) increase the rate and extent of digestion of fiber, (b) improve nitrogen availability (either by decreasing the rate and extent of degradation of feed protein or by improving microbial protein synthesis), (c) redirect the microbial fermentation to enhance the amounts or ratios of products that serve as precursors for milk or meat, and (d) detoxify feed or forage components.

Many species) Bacillus cereus Brucella abortus Clostridium perfringens Coxiella burnetii Leptospira spp. Mycobacterium bovis Serratia marcesens Prototheca zopfii (alga) Source: duPreez and Giesecke, 1994. lactiae was once the most common causative agent, but it has been displaced over the past few decades by Staphylococcus aureus. , appear to have a growing involvement as causative agents of mastitis, as does Listeria monocytogenes. Mastitic infection can occur via the blood or by trauma to the udder, but it far more commonly occurs via the streak canal of the teat.

1996). The involvement of microbes in bloat is controversial (Clarke and Reid, Microbiology of the Dairy Animal 33 1974). Microbes certainly are involved to the extent that the ruminal fermentation is responsible for production of methane and carbon dioxide gases and the acids that reduce the ruminal pH and cause release of carbon dioxide from the ruminal bicarbonate pool. More direct roles of individual species of bacteria and protozoa have been difficult to establish. However, microbial involvement is suggested by two lines of evidence: (a) bloat is routinely and effectively inhibited by controlled release of monensin into the rumen (Cameron and Malmo, 1993) and (b) complete switching of ruminal contents between fistulated cattle having a high or low susceptibility to bloat results in a change of susceptibility that is maintained for approximately 24 h before the animal’s natural susceptibility or resistance reasserts itself (Clarke and Reid, 1974).

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Applied Dairy Microbiology by Elmer H. Marth, James Steele


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