By Paolo Boffetta, Stefania Boccia, Visit Amazon's Carlo La Vecchia Page, search results, Learn about Author Central, Carlo La Vecchia,

ISBN-10: 3319050672

ISBN-13: 9783319050676

ISBN-10: 3319050680

ISBN-13: 9783319050683

A quickly advisor to melanoma Epidemiology is a perfect addition to Springer Briefs in melanoma examine. The short offers middle options in melanoma epidemiology and in addition supplies a photo of the epidemiology of seventeen human cancers. The short goals to provide-with quantitative focus-estimates of the worldwide burden of neoplasms, of contemporary and sure destiny tendencies, distribution, motives and techniques for prevention for significant teams of cancers. ultimately, the short will supply an outline of severals components that reason melanoma together with nutritional elements, tobacco smoking, weight problems and alcohol consumption.

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Extra resources for A Quick Guide to Cancer Epidemiology

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2013). 5–3. Its occurrence is correlated with the incidence of colon cancer but not stomach cancer. Adenocarcinomas account for approximately 50 % of neoplasms of the small intestine. They originate mainly in the duodenum and proximal jejunum and are preceded by formation of adenoma. Various hereditary syndromes such as familial adenomatous polyposis and Peutz-Jeghers syndrome, are characterized by multiple hamartomatous adenomas of the small intestine and, to a less extent, of the colon: these patients carry an increased risk of adenocarcinoma of the small intestine.

An additional role of gallbladder hypomotility in stone formation is likely. In Asia, the main types of gallstone are formed by bilirubin salts and have as risk factor bacterial infection of the biliary system: their association with gallbladder cancer, however, is not clear (Hsing et al. 2006). The increased rate of cholecystectomy in many high resource countries is probably responsible for the temporal decreasing trend of gallbladder cancer (Randi et al. 2006). Other suspected risk factors for gallbladder cancer include chronic inflammation, biliary stasis and infection, in particular status of chronic typhoid and paratyphoid carrier, history of gastric resection, reproductive history resulting in increased exposure to endogenous oestrogens and progesterone, obesity and, possibly, increased energy intake.

The risk in smokers is about twofold higher than that in non-smokers (Gandini et al. 2008), and a dose–response relationship and a favourable effect of quitting smoking have been shown in many populations. The proportion of cases of pancreatic cancer 34 4 Distribution, Causes and Prevention of Individual Neoplasms attributable to tobacco smoking has been estimated to be 15–20 % (Bosetti et al. 2012b). Some of the features of the descriptive epidemiology of pancreatic cancer (that is, a high incidence among Blacks in the USA as compared to a low incidence in Africa, and a higher risk among men and urban residents) can be explained by differences in smoking habits.

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A Quick Guide to Cancer Epidemiology by Paolo Boffetta, Stefania Boccia, Visit Amazon's Carlo La Vecchia Page, search results, Learn about Author Central, Carlo La Vecchia,

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